Host resistance - The gene for gene releationship

15 important questions on Host resistance - The gene for gene releationship

What is the resistance of the most plant effective against?

- The resistance of most plants is effective against certain genotypes of pathogen.
- The effectiveness depends on genes in both the plant and pathogen.

What is the gene-for-gene model?

- The gene-for-gene model hypothesizes a specific gene in the host plant for each gene in a pathogen conditioning for resistance.
- Emphasis on a gene for virulence in the pathogen and corresponding resistance in the host.

What is the concept behind the gene-for-gene hypothesis proposed by Flor?

- The gene-for-gene hypothesis suggests each gene for pathogen virulence has a corresponding gene for resistance in the host.
- Flor emphasized the interaction is specific to virulence genes.
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What does the gene-for-gene model assume about hypersensitive resistance?

- The model assumes hypersensitive resistance is activated by a gene when the corresponding avirulence gene is present in the pathogen.
- Resistance is specific to certain virulence genes.

What do R1 and R2 represent in the gene-for-gene hypothesis?

- R1 and R2 represent loci for hypersensitivity resistance in the host plant.
- These are specific for the corresponding avirulence genes.

How do R1 and R2 function according to the gene-for-gene hypothesis?

- R1 and R2 function by carrying alleles for resistance that correspond to specific avirulence genes in the pathogen.
- Resistance is effective only with matching avirulence genes.

What are the key concepts discussed in relation to plant-pathogen interactions?

- The interaction results in either compatibility or incompatibility.
- Resistance and susceptibility are terms used to describe plant reactions.
- Avirulence genes in pathogens are countered by plant proteins.
- Pathogens may produce necrotrophic toxins to promote pathogenesis.

What is accumulating in regard to the molecular basis of the gene-for-gene relationship?

- Dominant alleles for resistance are involved.
- Dominant alleles for avirulence lead to a specific recognition.
- This recognition triggers the hypersensitive response.

Where does recognition likely take place in the gene-for-gene relationship?

- Recognition occurs at the plant cell membrane or cytoplasm.
- It is based on whether a pathogen carries a functional avirulence gene.

What happens if the pathogen carries a mutated avirulence gene?

- No recognition occurs at the plant cell if the avirulence gene is mutated.
- No hypersensitivity response is elicited.
- The plant can be infected successfully by the pathogen.

What is avirulence gene specificity based on?

- Avirulence gene specificity is based on the effector inside the host plant cell.
- It is determined by the N-terminal and C-terminal domains.

What does the gene-for-gene hypothesis commonly assert?

- It asserts resistance is effective against all isolates of the pathogen.
- This assertion assumes a gene for avirulence corresponding to the resistance gene.

Why might a resistance gene not be race-specific according to the text?

- Resistance gene might not be race-specific if it functions against all pathogen isolates.
- Not race-specific when avirulence gene is not linked to a particular pathogen race.

What does the gene-for-gene relationship imply?

- This relationship implies differential interaction across pathogen strains.
- It involves ranked resistance levels and isolates' reaction amount.

What is the importance of reaction types in the gene-for-gene relationship?

- Reaction types demonstrate if major genes for resistance and avirulence occur.
- These genes are important for differential interaction.

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