Nuclear Hormone Receptors and metabolism
5 important questions on Nuclear Hormone Receptors and metabolism
How does the binding of estrogen to its receptor cause chromatin remodelling?
- Recruitment of co-activator
- acetylation of histone
- binding of the remodelling engine
- chromatin remodelling
- recruitment of RNA polymerase 2
This histone acetylation can be removed by Histone deacetylases (HDACs)
What is the difference between PPAR-y and PPAR-a?
- is active in adipose and uptake/store fats (agonist is Thiazolidinediones = TZDs)
PPAR-a
- is active in liver and burn fat (agonists is fibrates)
What does Cortisol (glucocorticoid hormone)?
- Released from adrenal cortex (bijnieren) in response to fasting and chronic stress
- bind nuclear receptor GR and modulate gene expression, among which enzymes involved in metabolism
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What are the 3 drug against Glucocorticoid?
- Hydrocortisone
- Prednisone
- Prednisolone
What are the long-term effects of cortisol
- Activates: lipolysis, protein degradation, Gluconeogenesis, Blood glucose levels, beta-Oxidation
- Inhibits: Protein synthesis
Leads to increase of insuline > glycogen synthesis and fat synthesis increase
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