Synapses - Neuronal plasticity - Long term potentiation
6 important questions on Synapses - Neuronal plasticity - Long term potentiation
What is the function of the AMPA receptor on synapses?
When it's activated the channels open up and Na+ will enter the cell and K+ will go out of the cell, to cause the cell to depolarise.
What does the NMDA receptor do when its activated?
What does the metabotropic glutamate receptor (mGluR) do?
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What is needed to produce more AMPA receptors in the postsynaptic membrane?
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How does long-term potentiation (LTP) take place?
- Action potential start in a presynaptic neuron.
- glutamate neurotransmitter is released in the cleft.
- The glutamate transmitters bind to the AMPA and NMDA receptors on the postsynaptic neuron.
- The AMPA receptors activates (Na+ in K+ out) which causes depolarisation
- More Na+ will cause release of Mg2+ and thus activation of NMDA receptor which causes the influx of Ca2+
- More AMPA receptors will be formed and stored in the membrane of post synaptic neuron and stronger connections are formed.
How does stimulation of synapses after firing of action potential causes long term depression?
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